|  Mediators of Inflammation

General information on the online edition:

Title:

Mediators of Inflammation

Publisher / Provider:

Hindawi
Search publishers open access policy in SHERPA/RoMEO

Subject(s):

Medicine

Category:

DOAJ Seal'The DOAJ Seal is awarded to journals that demonstrate best practice in open access publishing. Around 10% of journals indexed in DOAJ have been awarded the Seal.' | Indexed in DOAJ'The Directory of Open Access Journals (DOAJ) indexes and provides access to high quality, open access, peer-reviewed journals. All DOAJ journals are automatically tagged in the EZB.' | Mir@bel'Mir@bel is a French co-operative network. It comprises 102 institutions, including mainly academic libraries, research and educational institutions, as well as 21 publishing partners. Together they operate the online portal https://reseau-mirabel.info/, a French research platform for electronic journals. The portal includes bibliographic data and licence information for around 17,000 publications and is an important component of information services in the French library landscape. (as of 2024)' | Open Access'Open Access means that scientific literature and research data are freely accessible and can be reused to a certain extent, often through the use of Creative Commons licenses.'

Keyword(s):

Allgemeine medizinische Zeitschriften

Language(s):

English

E-ISSN(s):

1466-1861

P-ISSN(s):

0962-9351

ZDB-Number:

2008065-7

Fulltext online since:

Jg. 1 (1992)

Homepage(s):

http://www.hindawi.com/journals/mi/
https://www.ncbi.nlm.nih.gov/pmc/journals/324/

Type of Appearance:

Fulltext, online and print

Price for reading access:

free of charge

Access conditions:

Open Access

Annotation:

Bis 2004 bei Taylor & Francis.
Hinweis auf der Seite http://www.hindawi.com/journals/MI/open-access.html: 'MI is an Open Access journal.'
Whilst not neglecting any of the earlier know mediators such as histamine, bradykinin, prostaglandins, leukotrienes and PAF, Mediators of Inflammation focuses on research into cytokines (tumour necrosis factors, interleukins, interferons), biological response modifiers and the family of cell adhesion-promoting molecules.